Hair Loss Treatments & Clinics in Burleigh Heads, QLD
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3/34 Township Dr, Queensland, Burleigh Heads, 4220
0469381395
Thymoquinone (TQ), the main bioactive component of Nigella Sativa, has been found to exhibit anticancer effects in numerous preclinical studies. Due to its multi targeting nature, TQ interferes in a wide range of tumorigenic processes and counteracts carcinogenesis, malignant growth, invasion, migration, and angiogenesis. Moreover, TQ can specifically sensitize tumour cells toward conventional cancer treatments (e.g., radiotherapy, chemotherapy, and immunotherapy) and simultaneously minimize therapy-associated toxic effects in normal cells. In this review, we summarized the adjuvant potential of TQ as observed in various in vitro and in vivo animal models and discussed the pharmacological properties of TQ to rationalize its supplementary role in potentiating the efficacy of standard therapeutic modalities namely surgery, radiotherapy, chemotherapy, and immunotherapy. Altogether, we suggest further comprehensive evaluation of TQ in preclinical and clinical levels to delineate its implied utility as a novel complementary adjuvant therapy for cancer treatment. Introduction For the last five decades, large-scale “war on cancer” leads to significant progress in the development of new therapeutic options and more in-depth insights about various malignancies. Despite the emergence of numerous treatment strategies such as chemotherapeutic agents, small molecule inhibitors, specific gene, or protein targeting so-called smart drugs, and immunotherapies that are found to be effective in the treatment of some form of cancers (e.g., childhood leukaemia, human epidermal growth factor receptor 2-positive breast cancer), cancer death rate, in general, has not changed substantially (only 5% since 1950; Sung et al., 2012). According to Centres for Disease control and Prevention (CDC), cancer is the second leading cause of death in US (591, 699 deaths registered in 2015–2016), almost catching up heart diseases (https://www.